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📰 L'Alsace · 23/04/2022
<a href="https://news.google.com/rss/articles/CBMitwFBVV95cUxPZ0VUQnl3Yk1UeUpEZGNqa0pJdG0zenJ1RU9XWTJ2WnpOWF9qMUpSSFg5SkRNVHlnREFtVHZSZXF3Wk1yUnFta0xCMEtEamdkZFBhdTlxRGxrZGNrSXdFdzdJV1hCY2VjR05PTVE1SmpOYW1OdWZUd0tjZmFIaFNVVnlYdjN6djlibExQRWg3ZGpDRjVCbkRjZDBVbVJCd0c3dWh2ZEJ4Y1hYX1htS3FEVWF5YjdmUlU?oc
📰 Yahoo Actualités · 07/12/2021
<a href="https://news.google.com/rss/articles/CBMijgFBVV95cUxNNnRXdlJ2c0ZLaUJaaFBwYXRVamVUUWtmYXBWMF9kcDBIc29NdlEtSXBUS0Fzb0VXaTJWN181X011Z01jbVRsYlBwajl2RFR0ZC1zQTVEWEItc1ZDWFk3OFhxaDJGb1lzQlRIRFFEdkFZNEFPRjhUdGhEbUYweGpoaHBRY3gxcXlPdGxSUEV3?oc=5" target="_blank">Alsace : prison ferme pour un coupl
Journal of neurosurgery · 1999
✓ Tophaceous pseudogout is one of the rarest forms of crystal deposition disease, typically presenting as a destructive and invasive mass involving the temporomandibular joint or the infratemporal fossa region in the absence of any other articular manifestations. Previous cases have been assumed to be caused by calcium pyrophosphate dihydrate (CPPD) crystal deposition, based on finding weakly birefringent crystals in the involved tissues. The authors present the unique case of a 65-year-old woman with a destructive and invasive facial mass extending to the middle cranial fossa with microscopic and clinical features consistent with tophaceous pseudogout. High-resolution x-ray crystallographic powder diffraction and Fourier transformed infrared spectroscopy subsequently revealed that the crystals were composed of calcium hydroxyapatite without CPPD. The patient was later found to have primary hyperparathyroidism and mild hypercalcemia. This case demonstrates that tissue deposits of calcium hydroxyapatite can cause a destructive and invasive mass containing weakly birefringent crystals and raises the question of whether previous cases attributed to tophaceous pseudogout resulting from CPPD actually were composed of birefringent calcium hydroxyapatite.
The American journal of surgical pathology · 2024
Validated nonbiopsy methods to assure duodenal mucosal healing in celiac disease are lacking, yet ongoing mucosal injury is associated with anemia, osteoporosis, and lymphoma. Most providers utilize clinical data as surrogates of mucosal status to avoid additional esophagogastroduodenoscopy. The reliability of such surrogates to predict mucosal recovery has been incompletely evaluated. The aim of this study was to rigorously assess patterns of histologic mucosal recovery at follow-up in celiac disease and to correlate findings with clinical data. Gastrointestinal pathologists from 13 centers evaluated initial and follow-up duodenal biopsies from 181 celiac disease patients. Marsh scores and intraepithelial lymphocytes (IELs)/100 enterocytes were assessed blindly. Histology at follow-up was correlated with symptoms, immunoglobulin A anti–tissue transglutaminase titers and gluten-free diet adherence. Fifty-six/181 (31%) patients had persistent villous blunting and 46/181 (25%) patients had just persistently elevated IELs at follow-up, with only 79/181 (44%) patients having complete histologic remission. IEL normalization (82/181; 45%) lagged villous recovery (125/181;69%). In a minority of patients, villous blunting was limited to proximal duodenal biopsies. No correlation was found between Marsh scores and symptoms, normalization of immunoglobulin A anti–tissue transglutaminase serology, or diet adherence. Children showed greater recovery of Marsh score (P<0.001) and IELs (P<0.01) than adults. Persistent mucosal injury is common in celiac disease, with discordant villous/IEL normalization. Pathologist awareness of expected findings in celiac disease follow-up biopsies, including their frequent lack of correlation with clinical data, is important for patient management, and has implications for eligibility criteria for therapeutics currently in development.
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Journal of neurosurgery · 1999 · Case Reports
Grant GA, Wener MH, Yaziji H, Futran N, et al.
Journal of neurosurgery · 1999 · Case Reports
Grant GA, Wener MH, Yaziji H, Futran N, et al.
The American journal of surgical pathology · 2024 · Journal Article
Patel N, Leffler DA, Al-Toma A, Mulder CJ, et al.