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2 raisons identifiées
Praticien-chercheur
16 articles scientifiques publiés — formation continue solide
Référence presse grand public
Cité 10 fois dans les médias — pédagogie reconnue
✨ Génération du profil synthétique IA en cours…
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📰 lanouvellerepublique.fr · 12/02/2026
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📰 lanouvellerepublique.fr · 06/03/2023
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📰 lanouvellerepublique.fr · 21/12/2022
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📰 lanouvellerepublique.fr · 20/01/2021
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📰 Le Spécialiste · 09/12/2020
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📰 lanouvellerepublique.fr · 07/11/2020
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📰 lanouvellerepublique.fr · 21/02/2020
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📰 Capital.fr · 20/01/2016
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📰 France 24 · 06/10/2010
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📰 France 24 · 04/02/2008
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The Journal of pathology · 2006
AbstractMany autoimmune diseases including rheumatoid arthritis (RA), Sjögren's syndrome (SS) and systemic lupus erythematosus (SLE) occur much more frequently in women than in men. There is much evidence that oestrogen is the major cause of this gender difference. Interestingly, oestrogen relieves the symptoms of RA and SS but it exacerbates SLE. This contradictory effect of oestrogen on autoimmune diseases is not well understood. Most of the effects of oestrogen are mediated by two receptors: oestrogen receptor α and β (ERα and ERβ). To determine whether these contradictory effects of oestrogen relate to the involvement of distinct effects of the two ERs, we investigated expression of ERα and ERβ in human secondary lymphoid tissues. We observed that, in tonsils, ERβ is expressed in lymphocytes of germinal centres (GC) and the follicular mantle zone as well as in granulocytes, while ERα is expressed only in activated germinal centres but not in the follicular zone. ERβ is the predominant ER in human leucocytes from peripheral blood, spleen and in leucocytes infiltrating cancers in both males and females. In addition, in different human lymphoma cell lines including Hodgkin lymphoma, Burkitt lymphoma, and multiple myeloma, ERβ is abundant while ERα is not detectable. Our results indicate that ERβ is the predominant type of ER in mature lymphocytes. We suggest that ERα and ERβ have distinct roles in secondary lymphoid tissues and that further studies with ERβ‐specific agonists will help to elucidate the role of ERβ in these tissues. Copyright © 2005 Pathological Society of Great Britain and Ireland. Published by John Wiley & Sons, Ltd.
Spine deformity · 2022
Spine deformity · 2021
Source PubMed · Recherche par auteur (homonymes possibles, vérifier l'affiliation).
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Bouton D, Fedorak G, Oeffinger DJ, Gupta P, et al.
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Sharp J, Bouton D, Anabell L, Degan T, et al.
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Welborn MC, Bouton D, Baksh N, Degan T, et al.
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Welborn MC, Bouton D
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Chandrananth J, Hannan R, Bouton D, Raney E, et al.
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Lindsay SE, Bauer J, Bouton D, Do P, et al.
Spine deformity · 2022 · Journal Article
Bouton D, Fedorak G, Oeffinger DJ, Gupta P, et al.
Journal of pediatric orthopedics · 2025 · Journal Article
Welborn MC, Samdani AF, Szczodry M, Gupta P, et al.
Journal of the Pediatric Orthopaedic Society of North America · 2025 · Journal Article
Bryson X, Albarran M, Pham N, Salunga A, et al.
Spine deformity · 2025 · Journal Article
Tuason DA, Jabbouri SS, Bouton DJ, Johnson M, et al.
Spine deformity · 2025 · Journal Article
Tuason DA, Jabbouri SS, Bouton DJ, Johnson M, et al.
Spine deformity · 2021 · Journal Article
Bouton D, Karol L, Poppino K, Johnston C
Spine deformity · 2017 · Journal Article
Bouton D, Karol L, Poppino K, Johnston C
Instructional course lectures · 2016 · Journal Article
Bouton D, Ho CA, Abzug J, Brighton B, et al.
The Journal of pathology · 2006 · Journal Article
Shim GJ, Gherman D, Kim HJ, Omoto Y, et al.