Dr Geraldine Bart

Bibliographie

• Septic pubic Symphysitis: newest radiological, microbiological and outcome findings in a rare and difficult-to-treat infection

  2025

  ArticleClinical Microbiology and Infection

• Efficacy and safety of co-trimoxazole in device-related bone and joint infections: a CRIOGO multicentre case–control study

  2024

  ArticleJournal of Antimicrobial Chemotherapy

• 2023 French recommendations for diagnosing and managing prepatellar and olecranon septic bursitis

  2024

  ArticleJoint Bone Spine

• Association between radiographic and functional outcome in vertebral osteomyelitis SPONDIMMO, a 6-month prospective multicenter cohort

  2023

  ArticleJoint Bone Spine

• 2022 SPILF - Clinical Practice guidelines for the diagnosis and treatment of disco-vertebral infection in adults

  2023

  ArticleInfectious Diseases Now

• Management and outcome of native joint septic arthritis: a nationwide survey in French rheumatology departments, 2016–2017

  2022

  ArticleAnnals of the Rheumatic Diseases

• Spine immobilization and neurological outcome in vertebral osteomyelitis SPONDIMMO, a prospective multicentric cohort

  2022

  ArticleJoint Bone Spine

• MRI Features Associated With the Detection of Microbial Pathogens by CT-Guided Biopsy in Septic Spondylodiscitis

  2022

  ArticleJCR: Journal of Clinical Rheumatology

Source : HAL — archive ouverte CCSD/CNRS (couvre articles, chapitres EMC, communications congrès, thèses).

Localisation

Adresses géocodées via la Base Adresse Nationale (api-adresse.data.gouv.fr). Précision indicative.

Lieu de consultation

• HNIA PERCY

  101 Avenue HENRI BARBUSSE, 92140 Clamart

  ☎ 0141466000Hospitalier🏥 Voir cet établissement
  9 m12 m15 m16 m

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Tarifs & secteur de conventionnement

Secteur de conventionnement non disponible (médecin hospitalier ou non présent dans l'Annuaire santé CNAM des libéraux conventionnés).

Prendre rendez-vous & contact

Lien Doctolib = recherche Google site:doctolib.fr (le 1er résultat est presque toujours le profil correct s'il existe).

Top publications · les plus citées

• 1

  Distinct expression of interleukin (IL)-36α, β and γ, their antagonist IL-36Ra and IL-38 in psoriasis, rheumatoid arthritis and Crohn's disease

  Clinical and experimental immunology · 2016

  📚 236 citations🎯 RCR 8.77Top 3% NIH🔓 Open Access

  Lire l'abstract Crossref ↓

  Summary Interleukin (IL)-36α, IL-36β and IL-36γ are expressed highly in skin and are involved in the pathogenesis of psoriasis, while the antagonists IL-36Ra or IL-38, another potential IL-36 inhibitor, limit uncontrolled inflammation. The expression and role of IL-36 cytokines in rheumatoid arthritis (RA) and Crohn's disease (CD) is currently debated. Here, we observed that during imiquimod-induced mouse skin inflammation and in human psoriasis, expression of IL-36α, γ and IL-36Ra, but not IL-36β and IL-38 mRNA, was induced and correlated with IL-1β and T helper type 17 (Th17) cytokines (IL-17A, IL-22, IL-23, CCL20). In mice with collagen-induced arthritis and in the synovium of patients with RA, IL-36α, β, γ, IL-36Ra and IL-38 were all elevated and correlated with IL-1β, CCL3, CCL4 and macrophage colony-stimulating factor (M-CSF), but not with Th17 cytokines. In the colon of mice with dextran sulphate sodium-induced colitis and in patients with CD, only IL-36α, γ and IL-38 were induced at relatively low levels and correlated with IL-1β and IL-17A. We suggest that only a minor subgroup of patients with RA (17–29%) or CD (25%) had an elevated IL-36 agonists/antagonists ratio, versus 93% of patients with psoriasis. By immunohistochemistry, IL-36 cytokines were produced by various cell types in skin, synovium and colonic mucosa such as keratinocytes, CD68+ macrophages, dendritic/Langerhans cells and CD79α+ plasma cells. In primary cultures of monocytes or inflammatory macrophages (M1), IL-36β and IL-36Ra were produced constitutively, but IL-36α, γ and IL-38 were produced after lipopolysaccharide stimulation. These distinct expression profiles may help to explain why only subgroups of RA and CD patients have a potentially elevated IL-36 agonists/antagonists ratio.

• 2

  IL-38 overexpression induces anti-inflammatory effects in mice arthritis models and in human macrophages in vitro

  Annals of the rheumatic diseases · 2017

  📚 111 citations🎯 RCR 4.22Top 10% NIH
• 3

  2020 French recommendations on the management of septic arthritis in an adult native joint

  Joint bone spine · 2020

  📚 41 citations🎯 RCR 3.33Top 14% NIH🔓 Open Access📄 PDF gratuit ↗

Publications scientifiques (23) — classées par pathologie

Source PubMed · Recherche par auteur (homonymes possibles, vérifier l'affiliation).

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