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RhumatologueMédecins généralistes et spécialistes👤 Libéral intégral

M. Docteur Bernard MAILLET

📍 Moulins (03)Libéral💶 Secteur 2RPPS 10003143053
📊 Reconnaissance scientifique : 19/100📝 71 articles publiés📚 HAL (8)

Diplômes

🎓 DES & spécialité ordinale

  • Rhumatologie (SM)

📚 CES (Certificat d'Études Spéciales)

  • CES Rhumatologie

🎓 Diplômes

  • DE Docteur en médecine

Source : Annuaire Santé ANS (FHIR Practitioner.qualification) · Mises à jour quotidiennes.

Activité de recherche & publications

Source : bases de données publiques (OpenAlex, PubMed).

h-index

19

h articles cités ≥ h fois chacun. Un h de 19 = 19 publications avec 19+ citations.

Citations

2 196

Publications

71

i10-index

24

Thématiques principales

  • Pancreatitis Pathology and Treatment ×14
  • Osteoarthritis Treatment and Mechanisms ×12
  • Pancreatic and Hepatic Oncology Research ×10
  • Innovations in Medical Education ×6
  • Gastrointestinal disorders and treatments ×6

Affiliations FR : Saint Laurent Polyclinique · Société Française de Rhumatologie

Voir sur OpenAlex ↗

Source : OpenAlex (CC0, OurResearch). Indicateurs académiques agrégés sur 250 M+ d'œuvres.

Bibliographie

Source : HAL — archive ouverte CCSD/CNRS (couvre articles, chapitres EMC, communications congrès, thèses).

Localisation des cabinets

Adresses géocodées via la Base Adresse Nationale (api-adresse.data.gouv.fr). Précision indicative.

Lieux de consultation

Tarifs & secteur de conventionnement

🟡 Secteur 2 — Honoraires libresSource CNAM (Annuaire santé Ameli)
💳 Carte VitaleLibéral intégral
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Prendre rendez-vous & contact

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Lien Doctolib = recherche Google site:doctolib.fr (le 1er résultat est presque toujours le profil correct s'il existe).

Articles de presse (4)

Source : Google News (recherche par nom complet — homonymes possibles, vérifier le contenu).

Top publications · les plus citées

  • 1
    Non-alcoholic duct destructive chronic pancreatitis

    Gut · 1997

    📚 244 citations🎯 RCR 9.19Top 3% NIH🔓 Open Access📄 PDF gratuit ↗
    Lire l'abstract Crossref ↓

    Background —The pathology of non-alcoholic chronic pancreatitis has not yet been sufficiently studied. Aims —To identify the major changes of pancreatic tissue in patients surgically treated for non-alcoholic chronic pancreatitis. Patients —Pancreatectomy specimens from 12 patients with non-alcoholic chronic pancreatitis, including four patients with autoimmune or related diseases (Sjögren’s syndrome, primary sclerosing cholangitis, ulcerative colitis, and Crohn’s disease), were reviewed. Methods —Morphological changes were studied histologically and immunohistochemically (to type inflammatory cells) and compared with the pancreatic alterations found in 12 patients with alcoholic chronic pancreatitis. Results —In patients with non-alcoholic chronic pancreatitis, with or without associated autoimmune or related diseases, pancreatic inflammation particularly involved the ducts, commonly resulting in duct obstruction and occasionally duct destruction. None of these features was seen in alcoholic chronic pancreatitis which, however, showed pseudocysts and calcifications. Conclusion —The pancreatic changes in patients with non-alcoholic chronic pancreatitis clearly differ from those with alcoholic chronic pancreatitis. The term chronic duct destructive pancreatitis is suggested for this type of pancreatic disease.

  • 2
    Early decrease of serum biomarkers of type II collagen degradation (Coll2-1) and joint inflammation (Coll2-1 NO₂ ) by hyaluronic acid intra-articular injections in patients with knee osteoarthritis: a research study part of the Biovisco study

    Journal of orthopaedic research : official publication of the Orthopaedic Research Society · 2013

    📚 49 citations🎯 RCR 2.17Top 24% NIH🩺 Clinique
    Lire l'abstract Crossref ↓

    AbstractTo measure the evolution of the serum levels of specific Osteoarthritis (OA) biomarker, Coll2‐1 and Coll2‐1 NO2 in knee osteoarthritic patients after viscosupplementation (VS). Fifty‐one patients with unilateral symptomatic knee were recruited for this prospective open label study. They received three intra‐articular injections of 2 ml of hyaluronic acid (Hylan GF‐20) and were followed for 3 months. Walking pain was evaluated and serum samples were taken at each visit. Coll2‐1 and Coll2‐1 NO2 were measured in the serum using specific immunoassays. Variations over time of each parameter and predictive factor of response were studied. Forty‐five patients were analyzed. The serum concentrations of Coll2‐1 and Coll2‐1 NO2 were significantly higher in KL III/IV patients compared to KL I/II patients at baseline and decreased systematically over time after VS. Its effect was ever more pronounced in patients with KL III/IV. The serum concentration of Coll2‐1 was significantly lower at baseline in responders than in non‐responders. This study suggests a rapid slowdown of type II collagen degradation and joint inflammation after VS with Hylan G‐20 and gives additional information for the validation of accurate biomarkers for OA. The serum level of Coll2‐1 appeared to be a predictive factor for response to treatment. © 2013 Orthopaedic Research Society. Published by Wiley Periodicals, Inc. J Orthop Res 31: 901–907, 2013

  • 3
    Early effect of hyaluronic acid intra-articular injections on serum and urine biomarkers in patients with knee osteoarthritis: An open-label observational prospective study

    Journal of orthopaedic research : official publication of the Orthopaedic Research Society · 2012

    📚 34 citations🎯 RCR 1.50🩺 Clinique
    Lire l'abstract Crossref ↓

    AbstractThe aim of the study was to investigate the effect of hyaluronic acid (HA) intra articular injections (IA) on osteoarthritis (OA) biomarkers in patients with knee OA. Prospective open label study. Fifty‐one patients with unilateral symptomatic K‐OA received IA injections of 2mL of HA on days (D) 1, 7, 14 and were followed 3 months. At D‐15 patients were examined and X‐rays performed, to exclude patients with bilateral K‐OA, or those with more than three symptomatic OA joints. From 15 days (D‐15) before the first injection to D90 concomitant therapies were unchanged. Walking pain (WP) on VAS was obtained at each visit. Urine (U) and serum (S) samples were obtained at D‐15, D1, D30, and D90. S‐C2C, S‐Cartilage oligomeric matrix protein, S‐HA, S‐CS 846 epitope, S‐type II collagen propeptide, and U‐type II collagen C telopeptide (U‐CTX II/creatinin) were assayed. Predictive factors of response were analyzed using logistic regression. Correlations between variables were obtained using Spearman test. Forty‐five patients were analyzed. Between D‐15 and D1 there was no difference for any biomarkers At D1, WP (SD) was correlated with U‐CTX II/creat (p = 0.006). Between D1 and D90: U‐CTX II/creat decreased significantly. After adjustment for confounding variables there was a significant correlation between clinical response and U‐CTX II/creat variation. U‐CTX II and S‐HA at baseline were independently predictive of clinical response. This study showed that 90 days after HA IA injections, U‐CTX II levels significantly decrease compared to baseline, suggesting a slowdown of type II collagen degradation. © 2011 Orthopaedic Research Society Published by Wiley Periodicals, Inc. J Orthop Res 30:679–685, 2012

Publications scientifiques (9) — classées par pathologie

Source PubMed · Recherche par auteur (homonymes possibles, vérifier l'affiliation).

Transversal8

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Pharmacovigilance1

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