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Rhumatologue

Docteur JEAN SABATIER

RPPS 10003735544
📊 Reconnaissance scientifique : 51/100📝 341 articles publiés📚 HAL (3)

Diplômes

🎓 DES & spécialité ordinale

  • Rhumatologie (SM)

📚 CES (Certificat d'Études Spéciales)

  • CES Rhumatologie

🎓 Diplômes

  • DE Docteur en médecine

Source : Annuaire Santé ANS (FHIR Practitioner.qualification) · Mises à jour quotidiennes.

Activité de recherche & publications

Source : bases de données publiques (OpenAlex, PubMed).

h-index

51

h articles cités ≥ h fois chacun. Un h de 51 = 51 publications avec 51+ citations.

Citations

9 343

Publications

341

i10-index

184

Thématiques principales

  • Ion channel regulation and function ×104
  • Venomous Animal Envenomation and Studies ×103
  • Nicotinic Acetylcholine Receptors Study ×74
  • HIV Research and Treatment ×33
  • Antimicrobial Peptides and Activities ×26

Affiliations FR : Centre National de la Recherche Scientifique · Centre National de la Recherche Scientifique · Aix-Marseille Université

Voir sur OpenAlex ↗🆔 ORCID 0000-0002-9040-5647

Source : OpenAlex (CC0, OurResearch). Indicateurs académiques agrégés sur 250 M+ d'œuvres.

Bibliographie

Source : HAL — archive ouverte CCSD/CNRS (couvre articles, chapitres EMC, communications congrès, thèses).

Lieu de consultation

Tarifs & secteur de conventionnement

Secteur de conventionnement non disponible (médecin hospitalier ou non présent dans l'Annuaire santé CNAM des libéraux conventionnés).

📊 Comprendre le remboursement

Prendre rendez-vous & contact

🗓 Trouver sur Doctolib📇 Ajouter à mes contacts

Lien Doctolib = recherche Google site:doctolib.fr (le 1er résultat est presque toujours le profil correct s'il existe).

Articles de presse (1)

Source : Google News (recherche par nom complet — homonymes possibles, vérifier le contenu).

  • L’essor de la chirurgie vasculaire ambulatoire - Doctissimo

    📰 Doctissimo · 20/06/2017

    <a href="https://news.google.com/rss/articles/CBMinAFBVV95cUxPUE1XemVuUDd1aXFyMGJtODlTOGk2Slg4c1U5VWx1bVI5Z2l3SXpmOWdFWF9lLWVDTDh4MzlralEzR1pwa1l4U3V4MTZwQk44MnhDckZmdTdkdzNLZjRUc2oyZUJmQ0duYnV0RnI3aThyZzlBV2JZWWVvR3QtX3pIN0p6QzBtMDJ2QXoxaTFFZzdreXlqdVRzTW1vdmY?oc=5" target="_blank">L’essor de la ch

Top publications · les plus citées

  • 1
    Bone mineral acquisition during adolescence and early adulthood: a study in 574 healthy females 10-24 years of age

    Osteoporosis international : a journal established as result of cooperation between the European Foundation for Osteoporosis and the National Osteoporosis Foundation of the USA · 1996

    📚 169 citations🎯 RCR 6.50Top 5% NIH
  • 2
    Local and sustained delivery of 5-fluorouracil from biodegradable microspheres for the radiosensitization of malignant glioma: a randomized phase II trial

    Neurosurgery · 2005

    📚 79 citations🎯 RCR 2.11Top 25% NIH🩺 Clinique
  • 3
    The Renin-Angiotensin System: A Key Role in SARS-CoV-2-Induced COVID-19

    Molecules (Basel, Switzerland) · 2021

    📚 58 citations🎯 RCR 4.06Top 11% NIH🔓 Open Access📄 PDF gratuit ↗
    Lire l'abstract Crossref ↓

    The novel severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), the causative agent of coronavirus disease 2019 (COVID-19), was first identified in Eastern Asia (Wuhan, China) in December 2019. The virus then spread to Europe and across all continents where it has led to higher mortality and morbidity, and was declared as a pandemic by the World Health Organization (WHO) in March 2020. Recently, different vaccines have been produced and seem to be more or less effective in protecting from COVID-19. The renin–angiotensin system (RAS), an essential enzymatic cascade involved in maintaining blood pressure and electrolyte balance, is involved in the pathogenicity of COVID-19, since the angiotensin-converting enzyme II (ACE2) acts as the cellular receptor for SARS-CoV-2 in many human tissues and organs. In fact, the viral entrance promotes a downregulation of ACE2 followed by RAS balance dysregulation and an overactivation of the angiotensin II (Ang II)–angiotensin II type I receptor (AT1R) axis, which is characterized by a strong vasoconstriction and the induction of the profibrotic, proapoptotic and proinflammatory signalizations in the lungs and other organs. This mechanism features a massive cytokine storm, hypercoagulation, an acute respiratory distress syndrome (ARDS) and subsequent multiple organ damage. While all individuals are vulnerable to SARS-CoV-2, the disease outcome and severity differ among people and countries and depend on a dual interaction between the virus and the affected host. Many studies have already pointed out the importance of host genetic polymorphisms (especially in the RAS) as well as other related factors such age, gender, lifestyle and habits and underlying pathologies or comorbidities (diabetes and cardiovascular diseases) that could render individuals at higher risk of infection and pathogenicity. In this review, we explore the correlation between all these risk factors as well as how and why they could account for severe post-COVID-19 complications.

Publications scientifiques (41) — classées par pathologie

Source PubMed · Recherche par auteur (homonymes possibles, vérifier l'affiliation).

Transversal34

▼

Essai clinique4

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Vraie vie / RWE2

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Corticothérapie1

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IRM ostéo-articulaire1

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Pédiatrie1

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Revue générale1

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Datasets & protocoles partagés

Source : DataCite — DOIs pour datasets, logiciels, protocoles, registres patient. Hors articles (déjà couverts).

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