Docteur FRANCOIS JACQUOT

Bibliographie

• Electron and spin density distributions and magnetic anisotropy in mixed-valent diruthenium(V) tetracarboxylates assessed via crystallographic and theoretical analyses

  2026

  ArticleDalton Transactions

• Bilayer orthogonal ferromagnetism in CrTe2-based van der Waals system

  2025

  ArticleNature Communications

• Magnetic evolution of Cr₂Te₃ epitaxially grown on graphene with post-growth annealing

  2024

  ArticleApplied Physics Letters

• Epitaxial van der Waals heterostructures of Cr2Te3 on 2D materials

  2023

  ArticlePhysical Review Materials

• Large-scale epitaxy of two-dimensional van der Waals room-temperature ferromagnet Fe$_5$GeTe$_2$

  2022

  Articlenpj 2D Materials and Applications

• Spin Hall effect and spin absorption in ferromagnetic materials

  2021

  CongrèsSpintronics XIV

• Independence of the inverse spin Hall effect with the magnetic phase in thin NiCu films

  2020

  ArticlePhysical Review Letters

• Evidencing under-barrier phenomena in a Yb( iii ) SMM: a joint luminescence/neutron diffraction/SQUID study

  2019

  ArticleInorganic Chemistry Frontiers

Source : HAL — archive ouverte CCSD/CNRS (couvre articles, chapitres EMC, communications congrès, thèses).

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Top publications · les plus citées

• 1

  Lysophosphatidylcholine 16:0 mediates chronic joint pain associated to rheumatic diseases through acid-sensing ion channel 3

  Pain · 2022

  📚 28 citations🎯 RCR 2.88Top 17% NIH🔓 Open Access

  Lire l'abstract Crossref ↓

  Abstract Rheumatic diseases are often associated to debilitating chronic pain, which remains difficult to treat and requires new therapeutic strategies. We had previously identified lysophosphatidylcholine (LPC) in the synovial fluids from few patients and shown its effect as a positive modulator of acid-sensing ion channel 3 (ASIC3) able to induce acute cutaneous pain in rodents. However, the possible involvement of LPC in chronic joint pain remained completely unknown. Here, we show, from 2 independent cohorts of patients with painful rheumatic diseases, that the synovial fluid levels of LPC are significantly elevated, especially the LPC16:0 species, compared with postmortem control subjects. Moreover, LPC16:0 levels correlated with pain outcomes in a cohort of osteoarthritis patients. However, LPC16:0 do not appear to be the hallmark of a particular joint disease because similar levels are found in the synovial fluids of a second cohort of patients with various rheumatic diseases. The mechanism of action was next explored by developing a pathology-derived rodent model. Intra-articular injections of LPC16:0 is a triggering factor of chronic joint pain in both male and female mice, ultimately leading to persistent pain and anxiety-like behaviors. All these effects are dependent on ASIC3 channels, which drive sufficient peripheral inputs to generate spinal sensitization processes. This study brings evidences from mouse and human supporting a role for LPC16:0 via ASIC3 channels in chronic pain arising from joints, with potential implications for pain management in osteoarthritis and possibly across other rheumatic diseases.

• 2

  Balloon reduction and cement fixation in calcaneal articular fractures: a five-year experience

  International orthopaedics · 2013

  📚 19 citations🎯 RCR 1.29🔓 Open Access📄 PDF gratuit ↗
• 3

  [Articular forms of sarcoidosis]

  La semaine des hopitaux : organe fonde par l'Association d'enseignement medical des hopitaux de Paris · 1973

  📚 18 citations🎯 RCR 1.32

Publications scientifiques (16) — classées par pathologie

Source PubMed · Recherche par auteur (homonymes possibles, vérifier l'affiliation).

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